Cardiac arrest after spinal anesthesia
- Authors
- Eon, K.; Ok, S.-H.; Lee, S.H.; Sohn, J.-T.
- Issue Date
- Nov-2024
- Publisher
- Societa Editrice Universo
- Keywords
- cardiac arrest; lipid emulsion; spinal anesthesia; toxicity
- Citation
- Clinica Terapeutica, v.175, no.6, pp 387 - 388
- Pages
- 2
- Indexed
- SCOPUS
- Journal Title
- Clinica Terapeutica
- Volume
- 175
- Number
- 6
- Start Page
- 387
- End Page
- 388
- URI
- https://scholarworks.gnu.ac.kr/handle/sw.gnu/74991
- DOI
- 10.7417/CT.2024.5143
- ISSN
- 0009-9074
- Abstract
- This letter addresses potential cardiovascular changes following spinal anesthesia. High spinal anesthesia with a T4 sensory blockade can lead to decreased venous return, reduced right atrial pressure, and bradycardia. These effects result from diminished sympathetic activity, increased parasympathetic activity, and inhibition of cardiac accelerator fibers. In some cases, bradycardia following spinal anesthesia may progress to cardiac arrest. Therefore, patients receiving high spinal anesthesia should be monitored closely. © Società Editrice Universo (SEU)
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- There are no files associated with this item.
- Appears in
Collections - College of Medicine > Department of Medicine > Journal Articles

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