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Cardiac arrest after spinal anesthesia

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dc.contributor.authorEon, K.-
dc.contributor.authorOk, S.-H.-
dc.contributor.authorLee, S.H.-
dc.contributor.authorSohn, J.-T.-
dc.date.accessioned2024-12-10T07:30:13Z-
dc.date.available2024-12-10T07:30:13Z-
dc.date.issued2024-11-
dc.identifier.issn0009-9074-
dc.identifier.urihttps://scholarworks.gnu.ac.kr/handle/sw.gnu/74991-
dc.description.abstractThis letter addresses potential cardiovascular changes following spinal anesthesia. High spinal anesthesia with a T4 sensory blockade can lead to decreased venous return, reduced right atrial pressure, and bradycardia. These effects result from diminished sympathetic activity, increased parasympathetic activity, and inhibition of cardiac accelerator fibers. In some cases, bradycardia following spinal anesthesia may progress to cardiac arrest. Therefore, patients receiving high spinal anesthesia should be monitored closely. © Società Editrice Universo (SEU)-
dc.format.extent2-
dc.language영어-
dc.language.isoENG-
dc.publisherSocieta Editrice Universo-
dc.titleCardiac arrest after spinal anesthesia-
dc.typeArticle-
dc.publisher.location이탈리아-
dc.identifier.doi10.7417/CT.2024.5143-
dc.identifier.scopusid2-s2.0-85210363101-
dc.identifier.bibliographicCitationClinica Terapeutica, v.175, no.6, pp 387 - 388-
dc.citation.titleClinica Terapeutica-
dc.citation.volume175-
dc.citation.number6-
dc.citation.startPage387-
dc.citation.endPage388-
dc.type.docTypeArticle-
dc.description.isOpenAccessN-
dc.description.journalRegisteredClassscopus-
dc.subject.keywordAuthorcardiac arrest-
dc.subject.keywordAuthorlipid emulsion-
dc.subject.keywordAuthorspinal anesthesia-
dc.subject.keywordAuthortoxicity-
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