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Emodin, a Potent Anthraquinone Mitigates MPTP-Induced Parkinsons' Disease Pathology by Regulating Nrf2 and Its Downstream Targets: In Silico and In Vivo Approach (feb,10.1007/s12035-025-04762-3,2025)

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dc.contributor.authorJahan, Sarwat-
dc.contributor.authorIkram, Muhammad-
dc.contributor.authorSiraj, Sami-
dc.contributor.authorUllah, Shakir-
dc.contributor.authorZakria, Muhammad-
dc.contributor.authorAhmad, Nasir-
dc.date.accessioned2025-06-12T06:02:14Z-
dc.date.available2025-06-12T06:02:14Z-
dc.date.issued2025-09-
dc.identifier.issn0893-7648-
dc.identifier.issn1559-1182-
dc.identifier.urihttps://scholarworks.gnu.ac.kr/handle/sw.gnu/78739-
dc.description.abstractIn the original version of this article, the beta actin in Figure 3A is completely identical to 4A in the manuscript. The figures should have appeared as shown below. Corrected Figure 3 (Figure presented.) Corrected Figure 4 (Figure presented.) The original article has been corrected. © The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature 2025.-
dc.format.extent3-
dc.language영어-
dc.language.isoENG-
dc.publisherSpringer Nature-
dc.titleEmodin, a Potent Anthraquinone Mitigates MPTP-Induced Parkinsons' Disease Pathology by Regulating Nrf2 and Its Downstream Targets: In Silico and In Vivo Approach (feb,10.1007/s12035-025-04762-3,2025)-
dc.typeArticle-
dc.publisher.location미국-
dc.identifier.doi10.1007/s12035-025-05091-1-
dc.identifier.scopusid2-s2.0-105007109339-
dc.identifier.wosid001497683700001-
dc.identifier.bibliographicCitationMolecular Neurobiology, v.62, no.9, pp 12371 - 12373-
dc.citation.titleMolecular Neurobiology-
dc.citation.volume62-
dc.citation.number9-
dc.citation.startPage12371-
dc.citation.endPage12373-
dc.type.docTypeCorrection-
dc.description.isOpenAccessN-
dc.description.journalRegisteredClassscie-
dc.description.journalRegisteredClassscopus-
dc.relation.journalResearchAreaNeurosciences & Neurology-
dc.relation.journalWebOfScienceCategoryNeurosciences-
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